Osteosit: Perbedaan antara revisi

Konten dihapus Konten ditambahkan
ESCa (bicara | kontrib)
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Baris 46:
| accessdate = 2011-07-24
| work = Division of Anatomy, Department of Oral Growth and Development, Health Sciences University of Hokkaido School of Dentistry, Division of Anatomy and Cell Biology of the Hard Tissue, Department of Tissue Regeneration and Reconstruction, Niigata University Graduate School of Medical and Dental Sciences; Kazuharu Irie, Sadakazu Ejiri, Yasunori Sakakura, Toru Shibui, dan Toshihiko Yajima
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[[Simtoma]] [[hipoksia]] akan dengan cepat menginduksi [[kluster diferensiasi]] [[osteopontin]] pada osteosit melalui [[lintasan metabolisme|lintasan]] [[protein kinase C]], dan memicu [[osteoklastogenesis]] yang menghasilkan [[osteoklas]].<ref>{{en}}{{cite web
| url = http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1435734
| title = Upregulation of Osteopontin by Osteocytes Deprived of Mechanical Loading or Oxygen
| accessdate = 2011-07-24
| work = Orthopaedic Science Laboratories, Department of Orthopaedics and Sports Medicine, University of Washington; Ted S Gross, Katy A King, Natalia A Rabaia, Pranali Pathare, dan Sundar Srinivasan
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