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| work = Laboratory of Molecular Biology, Center for Cancer Research, National Cancer Institute, National Institutes of Health; Cheng SY, Leonard JL, Davis PJ.
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T(3) can activate phosphatidylinositol 3-kinase by a mechanism that may be cytoplasmic in origin or may begin at integrin alphavbeta3. Downstream consequences of phosphatidylinositol 3-kinase activation by T(3) include specific gene transcription and insertion of Na, K-ATPase in the plasma membrane and modulation of the activity of the ATPase.
 
== Membran mitokondria ==